By Kasugai, Yu; Machtoub, Lina
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Additional info for Amyotrophic lateral sclerosis : advances and perspectives of neuro-nanomedicine
Flachenecker P. Epidemiology of neuroimmunological diseases. J Neurol. 2006;253(suppl 5):V2–V8. 2. Mayeux R. Epidemiology of neurodegeneration. Annu Rev Neurosci. 2003;26:81–104. 3. Liu B, Hong JS. Role of microglia in inﬂammation-mediated neurodegenerative diseases: mechanisms and strategies for therapeutic intervention. J Pharmacol Exp Ther. 2003;304:1–7. 4. Yase Y. The pathogenesis of amyotrophic lateral sclerosis. Lancet. 1972;2:292–296. 5. Fang F, Kamel F, Lichtenstein P, et al. Familial aggregation of amyotrophic lateral sclerosis.
5 times more common than average within one-half mile of a lake or pond where cyanobacteria have bloomed. It has been hypothesized that people living around the lakes may have breathed in BMAA from the air, eaten ﬁsh contaminated with it, or accidentally swallowed it while swimming. Test were conducted of brain bank tissue to see if the ALS patients in these regions do in fact have elevated levels of BMAA. While the evidence mounts, researchers are already thinking about ways to detect toxic exposure before it causes disease.
Cognitive abnormalities associated with clinical Alzheimer’s disease as deﬁned by NINCDS-ADRDA criteria If these clinical ﬁndings occur, then close attention should be paid to the possible diagnosis of other disease processes. LMN and UMN signs may occur together with other clinical signs in disease where the pathologic process is not primary motor neuron degeneration. The physical and neurological examinations will allow for the clinical diagnosis of ALS to a particular degree of certainty as deﬁned above.
Amyotrophic lateral sclerosis : advances and perspectives of neuro-nanomedicine by Kasugai, Yu; Machtoub, Lina